Nutritional disorders of the nervous system

The central and peripheral nervous system may suffer deprivation of various nutrients. Pyridoxine (vitamin B6), thiamine (vitamin B1), cyanocobalamin (vitamin B12), Nicotinic Acid (vitamin B3), Pantothenic acid deficiencies are important among them.

Nutritional polyneuropathy: this may be the shortcomings of thiamine pyridoxine, cyanocobalamin, niacin, Pantothenic acid, alone or in combination.

Thiamin deficiency: this leads to neurological, cardiac or mal-efectos, is known as dry beriberi. Although originally described among people who subsist on rice polished as the main energy intake, the present beriberi is seen more among alcoholics. Neurologic injury takes the form of polyneuropathy symmetrical sensorimotor, characterized clinically by tingling, numbness and distal parts of the extremities.Motor phenomena are lower motor neuron bilateral lesions characterized by loss of two times ankle, gout and the fall of the wrist. There are deep, giving rise to the sensitivity of the calf hyperaesthesia. Some cases can develop Wernicke's encephalopathy.

Vitamin B12 deficiency resulting neuropathy can coexist with myelopathy in many cases.Neuropathy can be provable in 50-60% of subjects suffering from pellagra and subclinical deficiency of vitamin B6 deficiency niacina.La form produces polyneuropathy.More floridly is considered as a complication of isonicotinic acid (INH) hydrazide therapy.6 Mg of vitamin B6 administration prevents the development of this type of neuropathy.

Burning feet syndrome (also known as a condition in which the subject experience burning pain in the feet, especially Gopalan serious during the night, in order to interrupt the dream. examination may reveal cyanosis, excessive sweating, pallor, capillary dilation on both third pies.Una areas of these patients may show the sensibilidad.Mas loss often have may be presente.Muchos patients respond dramatically to injections of Pantothenic acid, suggesting that this vitamin deficiency can play an important role in the pathogenesis of the syndrome.)